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Sickle cell sickness benefits from a homozygous missense mutation within the β-globin gene that triggers polymerization of hemoglobin S. Gene therapy for patients with this problem is challenging by the advanced cellular abnormalities and troubles in attaining helpful, persistent inhibition of polymerization of hemoglobin S. We describe our 1st individual http://case-study-help27267.fireblogz.com/4477546/what-does-case-solution-mean


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